Skip to main content

Hashimotos – Autoimmune, Hypothyroid Triggers

What is Hashimoto’s?

Hashimoto’s thyroiditis is an autoimmune condition that is also known as, ‘Chronic Lymphocytic Thyroiditis’ or “Autoimmune Thyroiditis”. The “-itis” bit at the end of a body parts name indicates inflammation. The immune system creates antibodies against your own thyroid gland and attacks it as if it was a foreign body. This leads to Hypothyroid function.

The immune system attacks the thyroid gland and damages it, destroying the tissue that makes thyroid hormones. With enough damage and attack your thyroid gland gets less and less capable of making thyroid hormones and you go into “hypothyroidism” or being clinically diagnosed as “Hypothyroid” (slow/reduced thyroid activity) and if destruction continues you may not be able to make any measurable thyroid hormone at all.

Symptom Picture of Hashimoto’s

There is a common link occurrence where those with pre-existing autoimmune conditions fall more likely to also develop Hashimoto’s disease and surprisingly, women are around 8 times more likely to develop Hashimoto’s than men.

Symptoms of Hashimoto’s/Hypothyroid include but not limited to:

  • Tiredness and apathy.
  • Weight gain or weight loss that can’t be explained with calorie: calorie out.
  • Inability to handle cold.
  • Chronic joint and muscle pain throughout the day and especially on waking.
  • constipation.
  • Thin brittle and dry hair.
  • Irregularities in the menstrual cycle for women.
  • Lumpy cystic breast tissue (fibrocystic breast tissue).
  • low mood and depressive states.
  • Poor memory recall.
  • Very low heart rate especially at rest.

Links with the development of Autoimmune in the Thyroid.

Iodine deficiency or excess (1)

Iodine is a major component of thyroid hormones. Both extreme deficiency and excess can lead to autoimmune thyroiditis. Iodine deficiency, however, is more common and associated with more cases in general.
We need to consume between 90 to 290mcg of iodine per day for thyroid hormone production.

Iron Deficiency – Hypothyroid/Hashimotos 

Iron is in the center of an enzyme called TPO (thyroperoxidase) that builds the thyroid hormones inside the thyroid gland. Studies are now showing that a link between Iron deficiency impairs the thyroid metabolism. A common finding in pathology is relatively high levels of TSH with lower levels of T4 and in some cases goiter (swollen thyroid gland bulging from your neck usually linked to iodine deficiency) with adequate iodine.

Selenium Deficiency – Hypothyroid/Hashimotos 

It should be noted that not one single mineral can be used to correct or maintain the healthy function of the thyroid, however, one extremely important mineral involved in efficient thyroid function is Selenium. The enzyme that converts T4 to T3 is a selenoenzyme, meaning it is selenium dependant. Can’t work without selenium. In adults, per gram of tissue, the thyroid holds the most amount of selenium in the entire body.

Much of recent literature linking the deficiency of Selenium in adults that developed an autoimmune condition of the thyroid showed the correction of Selenium levels in the diet reduced Antithyroid antibody levels, improved the return ultrasound of the thyroid and improved overall quality of life. This is likely to be linked with selenium’s ability to aid in the regulation of our immune system response via its role in glutathione (GSH) production.

Take Home Message

It is imperative to understand as we mentioned earlier that, not one particular mineral, co-factor or nutrient source will directly correlate with the correction of thyroid function.

There are so many required factors in efficient thyroid activity and in the role of avoiding hypothyroid, so ensuring that you have adequate levels through diet, eliminate underlying drainage or overstimulation of the immune system by reducing known stressors, inflammation and low-grade inflammation triggering these immune responses as well is equally important. Both lifestyle and nutrients play equal pivotal roles. But remember you can get too much of a good thing. The thyroid gland running too fast will kill you so it will rebound and slow itself down if you are overdosing on thyroid nutrients.

Related Products:

The Development of T432 was with this in mind. Providing the tools required for thyroid metabolism be it in a current state of hyper or hypo, the nutrients provided will allow the body to correct the dysfunction itself. It just needs access to the tools, like a hammer needs a nail to complete the job.

With this, something like T432 and Resilience or ideally stacked with the 4 pillars (Multifood, Aurum oil, Resilience, and gutright) can really help in reducing immune and inflammatory triggers in their excess states, allowing the body to respond and return back to balance the way it is meant to. So if you haven’t already, there is a tonne of info on these for you to dig into! Find related articles below…

Related Articles:

T432 Plus

Episode 170 Autoimmune Diseases, Too much of a good thing

The Thyroid Clinical Picture of Hyperthyroidism or Hypothyroidism


  1. The relationship between autoimmune thyroid disease and iodine intake: a review. Endokrynol Pol. 1992;43 Suppl 1:53-69. Foley TP Jr1.
  2. Evidence suggesting that cadmium induces a non-thyroidal illness syndrome in the rat. J Endocrinol. 1997 Jul;154(1):113-7. Pavia Júnior MA1, Paier B, Noli MI, Hagmüller K, Zaninovich AA.
  3. Multiple nutritional factors and thyroid disease, with particular reference to autoimmune thyroid disease. Proc Nutr Soc. 2019 Feb;78(1):34-44. doi: 10.1017/S0029665118001192. Epub 2018 Sep 13. Rayman MP1.
  4. Should we treat subclinical hypothyroidism in obese children? BMJ. 2016 Mar 16;352:i941. doi: 10.1136/bmj.i941. Niranjan U1, Wright NP2.
  5. Iodine deficiency and thyroid disorders. Published:January 12, 2015DOI:  Prof Michael B Zimmermann, MD 

  6. Selenium and Human Health. Published:February 29, 2012DOI: Prof Margaret P Rayman, DPhil [Oxon] 
  7. Demographic and lifestyle factors and selenium levels in men and women in the U.S. Nutr Res Pract. 2011 Aug;5(4):357-64. doi: 10.4162/nrp.2011.5.4.357. Epub 2011 Aug 31. Park K1, Rimm E, Siscovick D, Spiegelman D, Morris JS, Mozaffarian D.
  8. Garber JR, Cobin RH, Garib H, et al. Clinical practice guidelines for hypothyroidism in adults: cosponsored by the American Association of Clinical Endocrinologists and the American Thyroid Association. Endocrine Practice. 2012;18(6):988–1028.

  9. Caturegli P, DeRemigis A, Rose NR. Hashimoto thyroiditis: clinical and diagnostic criteria. Autoimmunity Reviews. 2014;13(4-5):391–397.